Search results for "2406 Virology"

showing 5 items of 5 documents

Gamma interferon blocks gammaherpesvirus reactivation from latency in a cell type-specific manner

2007

Gammaherpesviruses are important pathogens whose lifelong survival in the host depends critically on their capacity to establish and reactivate from latency, processes regulated by both viral genes and the host immune response. Previous work has demonstrated that gamma interferon (IFN-gamma) is a key regulator of chronic infection with murine gammaherpesvirus 68 (gammaHV68), a virus that establishes latent infection in B lymphocytes, macrophages, and dendritic cells. In mice deficient in IFN-gamma or the IFN-gamma receptor, gammaHV68 gene expression is altered during chronic infection, and peritoneal cells explanted from these mice reactivate more efficiently ex vivo than cells derived from…

1109 Insect Sciencemedicine.medical_treatmentImmunologyCellSpleen610 Medicine & healthBiology10263 Institute of Experimental ImmunologyMicrobiologyInterferon-gammaGammaherpesvirinaeImmune systemVirologyVirus latencymedicineAnimalsHumansInterferon gammaDiphtheria toxinB-Lymphocytes2403 ImmunologyMacrophages2404 MicrobiologyHerpesviridae Infectionsmedicine.diseaseVirus LatencyCell biologyChronic infectionCytokinemedicine.anatomical_structureInsect ScienceImmunology2406 VirologyPathogenesis and Immunity570 Life sciences; biologyVirus Activationmedicine.drug
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Langerin+ DCs regulate innate IL-17 production in the oral mucosa during Candida albicans-mediated infection

2018

The opportunistic fungal pathogen Candida albicans frequently causes diseases such as oropharyngeal candidiasis (OPC) in immunocompromised individuals. Although it is well appreciated that the cytokine IL-17 is crucial for protective immunity against OPC, the cellular source and the regulation of this cytokine during infection are still a matter of debate. Here, we directly visualized IL-17 production in the tongue of experimentally infected mice, thereby demonstrating that this key cytokine is expressed by three complementary subsets of CD90+ leukocytes: RAG-dependent αβ and γδ T cells, as well as RAG-independent ILCs. To determine the regulation of IL-17 production at the onset of OPC, we…

Malemedicine.medical_treatment2405 ParasitologyPathology and Laboratory Medicine10263 Institute of Experimental ImmunologyMonocytesMice0302 clinical medicineAnimal CellsCandida albicansBiology (General)Candida albicansMononuclear Phagocyte SystemFungal PathogensInnate Immune Systemeducation.field_of_studyEukaryotaMononuclear phagocyte systemFlow CytometryCorpus albicans3. Good healthSpectrophotometryMedical MicrobiologyCytokinesCytophotometryCellular Types10244 Institute of VirologyQH301-705.5Immune CellsImmunologyMicrobiology03 medical and health sciences1311 GeneticsGenetics1312 Molecular BiologyeducationMicrobial PathogensMolecular BiologyMouth2403 ImmunologyBlood CellsOrganismsBiology and Life SciencesDendritic CellsMolecular DevelopmentYeastMice Inbred C57BLMannose-Binding Lectins030104 developmental biologyImmunologyThy-1 Antigens570 Life sciences; biologyParasitologyImmunologic diseases. AllergyDigestive SystemDevelopmental Biology0301 basic medicineNeutrophilsPhysiologyInterleukin-1betaYeast and Fungal ModelsInterleukin-23White Blood CellsSpectrum Analysis TechniquesCandidiasis OralImmune PhysiologyLeukocytesMedicine and Health SciencesCandidaStainingbiologyInterleukin-172404 MicrobiologyCell StainingSpecific Pathogen-Free OrganismsInfectious DiseasesCytokineExperimental Organism SystemsAntigens SurfaceFemaleAnatomyPathogensResearch ArticleLangerinPopulationMycologyOpportunistic InfectionsResearch and Analysis MethodsTongueImmunityVirologymedicineAnimalsLectins C-TypeInterleukin 6Interleukin-6Mouth MucosaFungiCell BiologyRC581-607biology.organism_classificationSpecimen Preparation and TreatmentImmune Systembiology.protein2406 VirologySpleen030215 immunology
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Host glycoprotein Gp96 and scavenger receptor SREC interact with PorB of disseminating Neisseria gonorrhoeae in an epithelial invasion pathway.

2007

Neisseria gonorrhoeae expresses numerous surface proteins that mediate bacterial adherence and invasion during infection. Gonococci expressing serotype A of the major outer membrane porin PorB (PorB(IA)) are frequently isolated from patients with severe disseminating infections. PorB(IA) triggers efficient adherence and invasion under low phosphate conditions mimicking systemic bloodstream infections. Here, we identify the human heat shock glycoprotein Gp96 and the scavenger receptor SREC as PorB(IA)-specific receptors. Gonococci expressing PorB(IA), but not those expressing PorB serotype B instead, bind to purified native or recombinant Gp96. Depletion of Gp96 from host cells prevented adh…

SerotypeCancer ResearchMICROBIO2405 ParasitologyPorinsBiologymedicine.disease_causeEndoplasmic ReticulumMicrobiologyBacterial Adhesionlaw.inventionMicrobiologyGonorrhealawVirologyImmunology and Microbiology(all)medicineAnimalsHumansScavenger receptorReceptorMolecular BiologyCells Culturedchemistry.chemical_classificationMembrane Glycoproteins10061 Institute of Molecular Cancer Research2404 MicrobiologyEpithelial CellsNeisseria gonorrhoeaeScavenger Receptors Class FchemistryPorin2406 VirologyRecombinant DNANeisseria gonorrhoeae570 Life sciences; biologyParasitologyGlycoproteinBacterial outer membraneProtein BindingCell hostmicrobe
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Anti-prothrombin autoantibodies enriched after infection with SARS-CoV-2 and influenced by strength of antibody response against SARS-CoV-2 proteins

2021

Antiphospholipid antibodies (aPL), assumed to cause antiphospholipid syndrome (APS), are notorious for their heterogeneity in targeting phospholipids and phospholipid-binding proteins. The persistent presence of Lupus anticoagulant and/or aPL against cardiolipin and/or β2-glycoprotein I have been shown to be independent risk factors for vascular thrombosis and pregnancy morbidity in APS. aPL production is thought to be triggered by–among other factors–viral infections, though infection-associated aPL have mostly been considered non-pathogenic. Recently, the potential pathogenicity of infection-associated aPL has gained momentum since an increasing number of patients infected with Severe Acu…

RNA virusesPulmonologyCoronavirusesPhysiology2405 ParasitologyAntibody Response030204 cardiovascular system & hematologyBiochemistrychemistry.chemical_compoundMedical Conditions0302 clinical medicineimmune system diseasesImmune PhysiologyMedicine and Health SciencesCardiolipinMedicineBiology (General)skin and connective tissue diseasesImmune ResponsePathology and laboratory medicineCOVIDVirus Testing0303 health sciencesLupus anticoagulantImmune System Proteinsbiologymedicine.diagnostic_test2404 MicrobiologyProteasesMedical microbiologyEnzymes3. Good healthInfectious DiseasesCoagulationVirusesSARS CoV 2PathogensAntibodyResearch ArticleSARS coronavirusQH301-705.5Immunology10208 Institute of Neuropathology610 Medicine & healthSARS-CoV-2; Respiratory infections; Virus testing; serine proteases; Antibody response; Autoantibodies; Blood plasma; ImmunoassaysResearch and Analysis MethodsMicrobiologyAntibodiesRespiratory Disorders03 medical and health sciences1311 GeneticsDiagnostic MedicineAntiphospholipid syndromeVirology1312 Molecular BiologyGeneticsImmunoassaysneoplasmsMolecular BiologyAutoantibodies030304 developmental biology030203 arthritis & rheumatology2403 ImmunologyPregnancyBiology and life sciencesbusiness.industryOrganismsViral pathogensAutoantibodyProteinsRC581-607medicine.diseaseMicrobial pathogenschemistry19ImmunoassayRespiratory InfectionsImmunology2406 VirologyEnzymologyImmunologic Techniquesbiology.protein570 Life sciences; biologyParasitologyImmunologic diseases. AllergySerine ProteasesbusinessPLOS Pathogens
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Guidelines for the Direct Detection ofAnaplasmaspp. in Diagnosis and Epidemiological Studies

2017

The genus Anaplasma (Rickettsiales: Anaplasmataceae) comprises obligate intracellular Gram-negative bacteria that are mainly transmitted by ticks, and currently includes six species: Anaplasma bovis, Anaplasma centrale, Anaplasma marginale, Anaplasma phagocytophilum, Anaplasma platys, and Anaplasma ovis. These have long been known as etiological agents of veterinary diseases that affect domestic and wild animals worldwide. A zoonotic role has been recognized for A. phagocytophilum, but other species can also be pathogenic for humans. Anaplasma infections are usually challenging to diagnose, clinically presenting with nonspecific symptoms that vary greatly depending on the agent involved, th…

10078 Institute of ParasitologyDirect diagnosis0301 basic medicineAnaplasma platysAnaplasmosisAnaplasmaAnaplasma bovisanimal diseases030231 tropical medicine610 Medicine & healthMicrobiology03 medical and health sciencesTicks0302 clinical medicine600 TechnologyZoonosesVirologyparasitic diseasesmedicineAnimalsHumansAnaplasmaInfecções Sistémicas e ZoonosesMicroscopybiologyIn vitro isolationAnaplasma ovis2404 Microbiology2725 Infectious DiseasesAnaplasma spp.bacterial infections and mycosesbiology.organism_classificationmedicine.diseaseVirologyAnaplasma phagocytophilumAnaplasmataceaePCR030104 developmental biologyInfectious DiseasesVertebrate hosts2406 Virology570 Life sciences; biologybacteriaAnaplasmosisRickettsialesVector-Borne and Zoonotic Diseases
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